Access this article Similar content being viewed by others Change history 21 May 2026 The original online version of this article was revised due to given name of the author Thuy Giang Nguyen Cao was incorrectly published as Thuy Nguyen Giang Cao
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Thirty-nine subjects completed the study (placebo, n = 19
Secondary flavoproteome defects associated with mitochondrial dysfunction Primary Mitochondrial Disorders Acyl-CoA dehydrogenase-9 deficiency (ACAD9) (OMIM #611126) Acyl-CoA dehydrogenase 9 (ACAD9), a mitochondrial protein, was initially demonstrated to catalyze the initial rate-limiting step in the beta-oxidation of long-chain fatty acids [48]
Even as the clinical community works to establish the long-term benefits of current GLP-1 and HRT combinations, a new generation of metabolic therapies is already moving through trials, ones that extend the biological logic of this article's central argument into territory that semaglutide and tirzepatide alone cannot reach
doi: 10.1210/jcem.83.7.4966 119 BellentaniSScaglioniFMarinoMBedogniG
